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FPR1 R123A
eagle-i ID
http://montana.eagle-i.net/i/00000131-0058-4b49-44b9-3efb80000000
Resource Type
cDNA plasmid
Properties
Construct Insert
FPR1 R123A
Resource Description
Human FPR1 with R123A point mutation. Point mutations of human FPR1 cDNA clone R-26 were generated by oligonucleotide-directed mutagenesis and confirmed by DNA sequencing.
Additional Name
fMet-Leu-Phe receptor; Formyl peptide receptor 1
Contact
Miettinen, Heini
Construct Backbone
pBGSA
Accession Number
M60627
Related Publication or Documentation
Formyl peptide receptor-mediated ERK1/2 activation occurs through G(i) and is not dependent on beta-arrestin1/2.
Related Publication or Documentation
Identification of putative sites of interaction between the human formyl peptide receptor and G protein.
Related Publication or Documentation
Activation and nuclear translocation of ERK1/2 by the formyl peptide receptor is regulated by G protein and is not dependent on beta-arrestin translocation or receptor endocytosis.
Selectable Marker
G418
Related Technique
Transfection
Location
Miettinen Lab
Inferred Types from the eagle-i Ontology (
What is an ontology?
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entity
continuant
independent_continuant
material_entity
Reagent
Construct
processed material
Plasmid
Thing
Provenance Metadata About This Resource Record
workflow state
Published
contributor
tjohnson
created
2011-07-06T11:48:29.221-05:00
creator
qking (Quinton King)
modified
2016-08-25T14:28:10.468-05:00